Search results for "Immunohistochemical Analysis"

showing 7 items of 7 documents

Bcl-2 as prognostic factor in head and neck squamous cell carcinoma

2005

A series of 66 cases of oral squamous cell carcinoma (OSCC) was retrospectively analyzed by immunohisto-chemistry for bcl-2 expression to verify its predictive value for clinical outcome in patients with OSCC. After grouping for bcl-2 expression, OSCCs were statistically analyzed for the variables age, gender, histological grading (G), TNM, staging, recurrence, and overall survival rate. Univariate and multivariate (Cox regression) analyses were performed. Thirty-six OSCC (54.5%) showed expression for bcl-2, whereas 30 (44.5%) were negative. No statistical association was found between bcl-2 expression and any variables considered at baseline. Overall disease-specific survival rate at 72 mo…

AdultMaleEXPRESSIONOncologyCancer Researchmedicine.medical_specialtyPathologyAdolescentSPONTANEOUS APOPTOSISPROTEINBCL-2Internal medicinemedicineCarcinomaHumansPROGNOSTIC SIGNIFICANCEGrading (tumors)Survival rateIMMUNOHISTOCHEMICAL ANALYSISORAL-CAVITYIN-VIVOAgedRetrospective StudiesAged 80 and overProportional hazards modelbusiness.industryCancerRetrospective cohort studyGeneral MedicineMiddle AgedPrognosismedicine.diseaseImmunohistochemistryCANCERHead and neck squamous-cell carcinomaSurvival RateFAMILY-MEMBERSstomatognathic diseasesProto-Oncogene Proteins c-bcl-2OncologySQUAMOUS CELL CARCINOMAHead and Neck NeoplasmsBAXCarcinoma Squamous CellRegression AnalysisImmunohistochemistryFemalebusinessONCOPROTEIN
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Association between COX-2 rs 6681231 genotype and interleukin-6 in periodontal connective tissue. A pilot study.

2014

[Objectives] The aim of this pilot study was to investigate associations between IL-6 and COX-2 expression in gingival biopsies and both clinical diagnosis and genotypes in the IL-6 and COX-2 genes. [Design] A case-control study included 41 gingival biopsies obtained from Caucasian patients grouped according to clinical diagnosis of gingival health (n = 10), gingivitis (n = 15) or chronic periodontitis (n = 16). Immunohistochemistry analyses were performed to determine COX-2 expression in lamina propria, IL-6 expression in lamina propria and gingival epithelium and level of inflammatory cell infiltrate. Individual DNA was extracted and genotyped by real-time PCR for IL6 SNPs rs 2069827 and …

Bacterial DiseasesMaleBiopsyGingivaDentistryGene ExpressionPilot ProjectsEpitheliumMonocytesGingivitisGenotypehealth care economics and organizationsPlasma cellsMultidisciplinaryGingival AbscessesbiologyQRMiddle AgedGingivitishumanitiesmedicine.anatomical_structureInfectious DiseasesCOX-2 6681231 genotype interleukin-6 periodontitisCytokinesPeriodontal AbscessesMedicineFemalemedicine.symptomPeriodontal IndexConnective tissueImmunohistochemical AnalysisResearch ArticleAdultmedicine.medical_specialtyClinical Research DesignScienceOral MedicineConnective tissueHemorrhagePolymorphism Single NucleotideInternal medicinemedicineGeneticsHumansInterleukin 6PeriodontitisBiologyAgedPeriodontitisClinical GeneticsInflammationbusiness.industryInterleukin-6Case-control studymedicine.diseaseChronic periodontitisHaplotypesCyclooxygenase 2Immune SystemCase-Control StudiesChronic Periodontitisbiology.proteinGenetic PolymorphismImmunologic TechniquesClinical ImmunologybusinessPopulation GeneticsPLoS ONE
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SNPs array karyotyping reveals a novel recurrent 20p13 amplification in primary myelofibrosis.

2011

The molecular pathogenesis of primary mielofibrosis (PMF) is still largely unknown. Recently, single-nucleotide polymorphism arrays (SNP-A) allowed for genome-wide profiling of copy-number alterations and acquired uniparental disomy (aUPD) at high-resolution. In this study we analyzed 20 PMF patients using the Genome-Wide Human SNP Array 6.0 in order to identify novel recurrent genomic abnormalities. We observed a complex karyotype in all cases, detecting all the previously reported lesions (del(5q), del(20q), del(13q), +8, aUPD at 9p24 and abnormalities on chromosome 1). In addition, we identified several novel cryptic lesions. In particular, we found a recurrent alteration involving cytob…

MaleMicroarraysMIELOFIBROSISChromosomes Human Pair 20Loss of Heterozygositylcsh:MedicineLoss of heterozygosityCohort StudiesHematologic Cancers and Related DisordersGene duplicationTaq Polymeraselcsh:ScienceOligonucleotide Array Sequence AnalysisMultidisciplinaryMYELOFIBROSIS; SNPKaryotypeGenomicsHematologyUniparental disomyMedicineFemaleImmunohistochemical AnalysisSNP arrayResearch ArticleTest Evaluationmedicine.medical_specialtyDNA Copy Number VariationsImmunologySNPLocus (genetics)Single-nucleotide polymorphismReceptors Cell SurfaceBiologyPolymorphism Single NucleotideDiagnostic MedicinemedicineGeneticsHumansBiologyAgedEvolutionary BiologyMyeloproliferative DisordersPopulation Biologylcsh:RCytogeneticsGene AmplificationComputational BiologyDNAUniparental Disomymedicine.diseaseMolecular biologyMYELOFIBROSISPrimary MyelofibrosisKaryotypingGenetic PolymorphismImmunologic TechniquesClinical Immunologylcsh:QPopulation GeneticsPLoS ONE
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Retinal ganglion cell loss is accompanied by antibody depositions and increased levels of microglia after immunization with retinal antigens.

2012

BackgroundAntibodies against retinal and optic nerve antigens are detectable in glaucoma patients. Recent studies using a model of experimental autoimmune glaucoma demonstrated that immunization with certain ocular antigens causes an immun-mediated retinal ganglion cell loss in rats.Methodology/principal findingsRats immunized with a retinal ganglion cell layer homogenate (RGA) had a reduced retinal ganglion cell density on retinal flatmounts (p = 0.007) and a lower number of Brn3(+) retinal ganglion cells (p = 0.0001) after six weeks. The autoreactive antibody development against retina and optic nerve was examined throughout the study. The levels of autoreactive antibodies continuously in…

MaleRetinal Ganglion Cellsgenetic structuresGlaucomaAutoimmunityImmune PrivilegeAutoantigenschemistry.chemical_compoundNeurobiology of Disease and RegenerationImmune ResponseMultidisciplinaryCell DeathMicrogliaQRAnimal ModelsImmunizationsmedicine.anatomical_structureNeurologyRetinal ganglion cellOptic nerveMedicineMicrogliaImmunohistochemical AnalysisResearch ArticleHistologyImmune CellsScienceImmunologyImmunoglobulinsModel OrganismsAntigenmedicineAnimalsAntibody-Producing CellsBiologyAutoantibodiesRetinabusiness.industryImmunityAutoantibodyGlaucomaRetinalbiochemical phenomena metabolism and nutritionmedicine.diseaseeye diseasesRatsOphthalmologychemistryRats Inbred LewImmunologyImmunologic TechniquesNeuro-OphthalmologyRatClinical ImmunologyImmunizationsense organsbusinessNeurosciencePLoS ONE
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Increased Vascularity in Cervicovaginal Mucosa with Schistosoma haematobium Infection

2011

Background Close to 800 million people in the world are at risk of schistosomiasis, 85 per cent of whom live in Africa. Recent studies have indicated that female genital schistosomiasis might increase the risk of human immunodeficiency virus (HIV) infection. The aim of this study is to quantify and analyse the characteristics of the vasculature surrounding Schistosoma haematobium ova in the female genital mucosa. Methodology/Principal Findings Cervicovaginal biopsies with S. haematobium ova (n = 20) and control biopsies (n = 69) were stained with immunohistochemical blood vessel markers CD31 and von Willebrand Factor (vWF), which stain endothelial cells in capillary buds and established blo…

PathologyAnatomy and PhysiologyBiopsyVDP::Medical disciplines: 700::Clinical medical disciplines: 750::Tropical medicine: 761Gynecologic InfectionsPathogenesisCervix UteriCardiovascularSchistosomiasis haematobiaVascularitySchistosomiasisSchistosoma haematobiumMicroscopyNeovascularization PathologicHistocytochemistrylcsh:Public aspects of medicineMucous membraneMiddle AgedImmunohistochemistryPlatelet Endothelial Cell Adhesion Molecule-1Infectious Diseasesmedicine.anatomical_structureMedical MicrobiologyVaginaSchistosoma haematobiumVaginaNeglected tropical diseasesMedicineFemalemedicine.symptomImmunohistochemical AnalysisResearch ArticleNeglected Tropical DiseasesAdultmedicine.medical_specialtylcsh:Arctic medicine. Tropical medicineHistologyAdolescentlcsh:RC955-962ImmunologySchistosomiasisBiologyMicrobiologyYoung AdultVascular Biologyvon Willebrand FactorParasitic DiseasesmedicineAnimalsHumansBiologySchistosomaMucous MembraneReproductive SystemParasite PhysiologyPublic Health Environmental and Occupational Healthlcsh:RA1-1270biology.organism_classificationmedicine.diseaseSchistosoma haematobium infectionAfricaImmunologic TechniquesWomen's HealthParasitologyGenital Diseases FemalePLoS Neglected Tropical Diseases
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Lung myofibroblasts are characterized by down-regulated cyclooxygenase-2 and its main metabolite, prostaglandin E2.

2013

Background: Prostaglandin E2 (PGE(2)), the main metabolite of cyclooxygenase (COX), is a well-known anti-fibrotic agent. Moreover, myofibroblasts expressing alpha-smooth muscle actin (alpha-SMA), fibroblast expansion and epithelial-mesenchymal transition (EMT) are critical to the pathogenesis of idiopathic pulmonary fibrosis (IPF). Our aim was to investigate the expression of COX-2 and PGE(2) in human lung myofibroblasts and establish whether fibroblast-myofibroblast transition (FMT) and EMT are associated with COX-2 and PGE(2) down-regulation. Methods: Fibroblasts obtained from IPF patients (n = 6) and patients undergoing spontaneous pneumothorax (control, n = 6) and alveolar epithelial ce…

PathologyPulmonologyMetaboliteImmunofluorescencelcsh:MedicineBiochemistrychemistry.chemical_compoundIdiopathic pulmonary fibrosisMolecular Cell BiologyPulmonary fibrosisProstaglandin E2Myofibroblastslcsh:ScienceLungCells CulturedFisiologia cel·lularMultidisciplinarybiologyFibrosi pulmonarrespiratory systemExtracellular Matrixmedicine.anatomical_structureCytokinesMedicinelipids (amino acids peptides and proteins)Immunohistochemical AnalysisMyofibroblastResearch ArticleSignal Transductionmedicine.drugmedicine.medical_specialtyEpithelial-Mesenchymal TransitionImmunologyInterstitial Lung DiseasesDinoprostonePulmonary fibrosisTransforming Growth Factor beta1ImmunofluorescènciaGrowth FactorsCell Line TumormedicineHumansEpithelial–mesenchymal transitionFibroblastBiologyCell Proliferationlcsh:RProteinsEpithelial Cellsmedicine.diseaseActinsIdiopathic Pulmonary Fibrosisrespiratory tract diseasesGene Expression RegulationchemistryCyclooxygenase 2Immune SystemCase-Control StudiesImmunologic Techniquesbiology.proteinCancer researchClinical Immunologylcsh:QCyclooxygenaseBiomarkersPLoS ONE
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Ovine Carotid Artery-Derived Cells as an Optimized Supportive Cell Layer in 2-D Capillary Network Assays

2014

PLoS one 9(3), e91664 (2014). doi:10.1371/journal.pone.0091664

Vascular Endothelial Growth Factor APathologyCellBecaplerminlcsh:MedicineCardiovascularUmbilical veinUmbilical CordDrug DiscoveryMolecular Cell BiologyBiological Systems EngineeringMyocyteCardiovascular Imaginglcsh:ScienceMultidisciplinaryProto-Oncogene Proteins c-sisAnimal ModelsFlow CytometryEndothelial stem cellBevacizumabmedicine.anatomical_structureCarotid ArteriesMonoclonalMedicineImmunohistochemical AnalysisResearch ArticleBiotechnologymedicine.medical_specialtyCell typeDrugs and DevicesDrug Research and DevelopmentMyocytes Smooth MuscleImmunologyBiomedical EngineeringBioengineeringBiologyAntibodies Monoclonal HumanizedCell LineModel OrganismsVascular Biologymedicine.arterymedicineAnimalsHumansBiologySheeplcsh:REndothelial CellsFeeder CellsUmbilical arteryMolecular biologyVascular Endothelial Growth Factor Receptor-2Coculture TechniquesCapillariesCell cultureImmunologic Techniqueslcsh:QCytometry
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